How does HCA work?
A. At the cellular level, HCA molecules “trick” the citrate lyase enzyme. The receptor sites of citrate lyase enzymes are filled with HCA. This means fewer fatty acids are synthesized, making less fat available for cellular storage. This results in less body fat build-up in the liver and adipose (fat) tissue. Increased amounts of energy at the cellular level are then available to more rapidly degrade free fatty acids. HCA suppresses appetite thus reducing food intake. When you consume food, the body begins breaking down carbohydrates into glucose (a necessary form of sugar). The liver functions to ensure that the blood continually has adequate glucose to fuel the brain with energy. Excess glucose molecules would normally break down into fat or cholesterol. However, (-)HCA temporarily inhibits the citrate lyase enzyme and excess glucose is converted to glycogen in the liver. When glucose and glycogen reach a certain level, the brain gets a signal of “fullness”, suppressing appetite and
